3-MethylindoleCAS# 83-34-1 |
Quality Control & MSDS
Number of papers citing our products
Chemical structure
| Cas No. | 83-34-1 | SDF | Download SDF |
| PubChem ID | N/A | Appearance | Powder |
| Formula | C9H9N | M.Wt | 131.1 |
| Type of Compound | Alkaloids | Storage | Desiccate at -20°C |
| Solubility | Soluble in Chloroform,Dichloromethane,Ethyl Acetate,DMSO,Acetone,etc. | ||
| General tips | For obtaining a higher solubility , please warm the tube at 37 ℃ and shake it in the ultrasonic bath for a while.Stock solution can be stored below -20℃ for several months. We recommend that you prepare and use the solution on the same day. However, if the test schedule requires, the stock solutions can be prepared in advance, and the stock solution must be sealed and stored below -20℃. In general, the stock solution can be kept for several months. Before use, we recommend that you leave the vial at room temperature for at least an hour before opening it. |
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| About Packaging | 1. The packaging of the product may be reversed during transportation, cause the high purity compounds to adhere to the neck or cap of the vial.Take the vail out of its packaging and shake gently until the compounds fall to the bottom of the vial. 2. For liquid products, please centrifuge at 500xg to gather the liquid to the bottom of the vial. 3. Try to avoid loss or contamination during the experiment. |
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| Shipping Condition | Packaging according to customer requirements(5mg, 10mg, 20mg and more). Ship via FedEx, DHL, UPS, EMS or other couriers with RT, or blue ice upon request. | ||
| Description | 3-Methylindole , a selective pneumotoxin, causes acute pulmonary edema and emphysema. | |||||
3-Methylindole Dilution Calculator
3-Methylindole Molarity Calculator
| 1 mg | 5 mg | 10 mg | 20 mg | 25 mg | |
| 1 mM | 7.6278 mL | 38.1388 mL | 76.2777 mL | 152.5553 mL | 190.6941 mL |
| 5 mM | 1.5256 mL | 7.6278 mL | 15.2555 mL | 30.5111 mL | 38.1388 mL |
| 10 mM | 0.7628 mL | 3.8139 mL | 7.6278 mL | 15.2555 mL | 19.0694 mL |
| 50 mM | 0.1526 mL | 0.7628 mL | 1.5256 mL | 3.0511 mL | 3.8139 mL |
| 100 mM | 0.0763 mL | 0.3814 mL | 0.7628 mL | 1.5256 mL | 1.9069 mL |
| * Note: If you are in the process of experiment, it's necessary to make the dilution ratios of the samples. The dilution data above is only for reference. Normally, it's can get a better solubility within lower of Concentrations. | |||||
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ClO2 pre-oxidation impacts the formation and nitrogen origins of dichloroacetonitrile and dichloroacetamide during subsequent chloramination.[Pubmed:32841932]
Water Res. 2020 Nov 1;186:116313.
Chlorine dioxide (ClO2) can be used as a pre-oxidant when chloramination is performed in water treatment plants. However, the effects of ClO2 pre-oxidation on the formation of nitrogenous disinfection by-products, such as dichloroacetonitrile (DCAN) and dichloroacetamide (DCAcAm), during chloramination are not well understood. In this study, the effects of ClO2 pre-oxidation on the formation of DCAN and DCAcAm during chloramination of 28 model compounds and seven real water samples were investigated. The sources of nitrogen for DCAN and DCAcAm formation were investigated using (15)N-labeled monochloramine. ClO2 pre-oxidation affected DCAN and DCAcAm formation during chloramination of model compounds in different ways. ClO2 pre-oxidation increased unlabeled and (15)N-labeled DCAN and DCAcAm formation during chloramination of six amino acids and peptides and five indoles and tertiary amines. ClO2 pre-oxidation decreased DCAN formation but increased DCAcAm formation during chloramination of three hydroxybenzamide compounds, but had the opposite effects for four tetracyclines. ClO2 pre-oxidation generally decreased DCAN and DCAcAm formation during chloramination of the phenolic compounds that are precursors not containing nitrogen. 2-Aminoacetophenone, formamid-trans-muconic acid, and unsaturated ketones were found to be transformation products of ClO2 oxidation of 3-Methylindole, salicylamide, and resorcinol, respectively. Possible DCAN and DCAcAm formation pathways during chloramination after ClO2 oxidation were identified. For most of the water samples, ClO2 pre-oxidation decreased the amounts of DCAN and DCAcAm formed during chloramination by 36%-70% and 11%-59%, respectively. This may have been caused by ClO2 oxidation destroying phenolic precursors and macromolecular proteins rather than amino acids in the water samples.
