Bisphenol PCAS# 2167-51-3 |
Quality Control & MSDS
Number of papers citing our products
Chemical structure
3D structure
| Cas No. | 2167-51-3 | SDF | Download SDF |
| PubChem ID | 630355 | Appearance | Powder |
| Formula | C24H26O2 | M.Wt | 346.5 |
| Type of Compound | N/A | Storage | Desiccate at -20°C |
| Solubility | Soluble in Chloroform,Dichloromethane,Ethyl Acetate,DMSO,Acetone,etc. | ||
| Chemical Name | 4-[2-[4-[2-(4-hydroxyphenyl)propan-2-yl]phenyl]propan-2-yl]phenol | ||
| SMILES | CC(C)(C1=CC=C(C=C1)C(C)(C)C2=CC=C(C=C2)O)C3=CC=C(C=C3)O | ||
| Standard InChIKey | GIXXQTYGFOHYPT-UHFFFAOYSA-N | ||
| Standard InChI | InChI=1S/C24H26O2/c1-23(2,19-9-13-21(25)14-10-19)17-5-7-18(8-6-17)24(3,4)20-11-15-22(26)16-12-20/h5-16,25-26H,1-4H3 | ||
| General tips | For obtaining a higher solubility , please warm the tube at 37 ℃ and shake it in the ultrasonic bath for a while.Stock solution can be stored below -20℃ for several months. We recommend that you prepare and use the solution on the same day. However, if the test schedule requires, the stock solutions can be prepared in advance, and the stock solution must be sealed and stored below -20℃. In general, the stock solution can be kept for several months. Before use, we recommend that you leave the vial at room temperature for at least an hour before opening it. |
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| About Packaging | 1. The packaging of the product may be reversed during transportation, cause the high purity compounds to adhere to the neck or cap of the vial.Take the vail out of its packaging and shake gently until the compounds fall to the bottom of the vial. 2. For liquid products, please centrifuge at 500xg to gather the liquid to the bottom of the vial. 3. Try to avoid loss or contamination during the experiment. |
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| Shipping Condition | Packaging according to customer requirements(5mg, 10mg, 20mg and more). Ship via FedEx, DHL, UPS, EMS or other couriers with RT, or blue ice upon request. | ||
Bisphenol P Dilution Calculator
Bisphenol P Molarity Calculator
| 1 mg | 5 mg | 10 mg | 20 mg | 25 mg | |
| 1 mM | 2.886 mL | 14.43 mL | 28.86 mL | 57.7201 mL | 72.1501 mL |
| 5 mM | 0.5772 mL | 2.886 mL | 5.772 mL | 11.544 mL | 14.43 mL |
| 10 mM | 0.2886 mL | 1.443 mL | 2.886 mL | 5.772 mL | 7.215 mL |
| 50 mM | 0.0577 mL | 0.2886 mL | 0.5772 mL | 1.1544 mL | 1.443 mL |
| 100 mM | 0.0289 mL | 0.1443 mL | 0.2886 mL | 0.5772 mL | 0.7215 mL |
| * Note: If you are in the process of experiment, it's necessary to make the dilution ratios of the samples. The dilution data above is only for reference. Normally, it's can get a better solubility within lower of Concentrations. | |||||
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Bisphenol P activates hormonal genes and introduces developmental outcomes in Chironomus tentans.[Pubmed:30878007]
Ecotoxicol Environ Saf. 2019 Jun 15;174:675-682.
The endocrine disrupting properties of bisphenol A (BPA) discharged to the environment have been newly identified by the European Chemicals Agency, increasing the need to assess the environmental endocrine disrupting potentials of its alternatives with which it shares close structural features. However, few investigations of the environmental endocrine disrupting functions of BPA analogs have been conducted to date. In this study, the endocrine disrupting effects of a BPA analog of Bisphenol P (BPP) were investigated in the nonbiting midge (Chironomus tentans), a model organism in ecotoxicology. An initial ex vivo test using salivary gland cells explanted from the larvae and a subsequent in vivo test using embryos and larvae revealed the upregulatory effects of BPP on ecdysone receptor genes encoding the ecdysone receptor (EcR) and the early responsive gene E74, with a similar temporal pattern of gene activation. Partial life cycle and full life cycle toxicity tests demonstrated BPP altered embryo hatching, larval emergence, and adult sex ratio at concentrations close to the effective concentrations for hormonal genetic endpoints in embryos and larvae after 48h of exposure. Although embryos appeared to be more sensitive to BPP than the fourth instar larvae, the impact on neither life stage seemed enough to estimate the developmental impairment of the insects. These results demonstrate the ecdysone pathway is a target of BPP, and that long-term exposure could cause apical effects on the development of C. tentans. The endocrine disrupting effects towards aquatic organisms, as well as the high persistence and bioconcentration potential, indicate an urgent need to assess the environmental risks associated with BPP.
